Chronic kidney disease (CKD) is classically and ineluctably at the origin of a syndrome associating mineral and bone laboratory abnormalities, cardiovascular disease, and clinical and histological bone disease.1 This syndrome, called CKD—mineral and bone disease (MBD), begins in the early stages of CKD and becomes overtly expressed during the dialysis period, persisting even after a successful kidney transplantation (KT). Historically called renal osteodystrophy (ROD), the CKD-MBD has now been separated into 3 main types of bone profiles, based on histomorphometry criteria of bone turnover, mineralization, and volume.